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Study summary · NAD⁺, sirtuins & cellular energy

Can exogenous ubiquinol increase plasma CoQ10 levels and improve mitochondrial function in patients with severe sepsis or septic shock?

In one paragraph

Ubiquinol, a randomized, double-blind, placebo-controlled trial in 38 adult patients (Crit Care 2015, PMID 26130237) — oral ubiquinol can increase CoQ10 levels in septic patients, but this pilot study did not show improvements in mitochondrial function or clinical outcomes. No differences were observed between the two groups for any of the secondary outcomes, including vascular endothelial biomarkers, inflammatory biomarkers, mitochondrial injury biomarkers, or clinical outcomes.

Source: Crit Care 2015, PMID 26130237 ↗ · Research map · How Magellan grades evidence · evidence confidence: low

Crit Care · 2015 · PMID 26130237 · DOI 10.1186/s13054-015-0989-3

Plain-English summary written and published by Magellan Longevity · medical review by Gabriel Radu, DO (physiatrist, NPI 1376861765). We summarize what the paper reported — we did not run this study.

The takeaway

Oral ubiquinol can increase CoQ10 levels in septic patients, but this pilot study did not show improvements in mitochondrial function or clinical outcomes.

The question

Can exogenous ubiquinol increase plasma CoQ10 levels and improve mitochondrial function in patients with severe sepsis or septic shock?

What they tested

The provision of exogenous ubiquinol will increase plasma CoQ10 levels and improve mitochondrial function in patients with severe sepsis or septic shock, given previous findings of decreased CoQ10 in septic shock.

How they did it

This was a randomized, double-blind, placebo-controlled pilot trial conducted at a single hospital. 38 adult patients (19 in each group) with severe sepsis or septic shock received either 200 mg enteral ubiquinol or placebo twice daily for up to seven days. Blood samples were collected at baseline, 12, 24, 48, and 72 hours to measure CoQ10 parameters (total, relative to cholesterol, oxidized, and reduced CoQ10) as the primary outcome, and secondary outcomes included vascular endothelial, inflammatory, and mitochondrial injury biomarkers, as well as clinical outcomes. Repeated measures models were used for group comparisons.

What they found

The ubiquinol group showed a significant increase in total CoQ10 levels, CoQ10 levels relative to cholesterol, and levels of oxidized and reduced CoQ10 compared to the placebo group. No differences were observed between the two groups for any of the secondary outcomes, including vascular endothelial biomarkers, inflammatory biomarkers, mitochondrial injury biomarkers, or clinical outcomes.

What it means

Oral ubiquinol administration successfully increased plasma CoQ10 levels in patients with severe sepsis or septic shock. However, this pilot trial did not demonstrate an improvement in mitochondrial function or clinical outcomes.

Limitations

This was a pilot trial, and further research is needed to determine if ubiquinol administration can lead to improved clinical outcomes in this patient population.

“significant increase in total CoQ10 levels in the ubiquinol group compared to placebo”— from the published abstract, Crit Care 2015

The paper at a glance

TitleUbiquinol in patients with severe sepsis or septic shock: a randomized, double-blind, placebo-controlled pilot trial
JournalCrit Care
Year2015
PMID26130237 ↗
DOI10.1186/s13054-015-0989-3 ↗
TopicNAD⁺, sirtuins & cellular energy

Read the source: PubMed record (authors, abstract, full citation) ↗ · Publisher via doi.org ↗

Where Magellan uses this paper

This citation sits behind the evidence grade on the pages below. Grades are set from the research and are independent of affiliate commissions.

Ubiquinol (Reduced CoQ10)

In human trials CoQ10 has been studied mainly as an adjunct in cardiovascular disease: the Q-SYMBIO randomized trial reported roughly halved major adverse cardiovascular events in chronic heart failure, and a 2024 meta-analysis of 33 heart-failure trials found lower all-cause mortality (RR 0.64) and hospitalization…

Ubiquinol (Reduced CoQ10)

Coenzyme Q10 (CoQ10, ubiquinone) is a fat-soluble compound the body synthesizes and also obtains in…

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Research describes the compound or intervention studied. It is not a claim about any specific product.

Molecules & mechanisms in this paper

Each of these is named in the paper’s own words above. Open the monograph for the full mechanism and its other citations.

Cite this page

These citations point at this summary. To cite the original paper with its full author list, use the PubMed record or doi.org.

APA
Magellan Longevity. (2026). Can exogenous ubiquinol increase plasma CoQ10 levels and improve mitochondrial function in patients with severe sepsis or septic shock? [Plain-English summary of Crit Care 2015, PMID 26130237, DOI 10.1186/s13054-015-0989-3]. Magellan Longevity. https://magellanlongevity.com/study/d26130237.html
BibTeX
@misc{magellan_d26130237, title = {Can exogenous ubiquinol increase plasma CoQ10 levels and improve mitochondrial function in patients with severe sepsis or septic shock?}, author = {{Magellan Longevity}}, year = {2026}, howpublished = {\url{https://magellanlongevity.com/study/d26130237.html}}, note = {Plain-English summary of PubMed PMID 26130237; DOI 10.1186/s13054-015-0989-3; Crit Care 2015. Reviewed by Gabriel Radu, DO}, urldate = {2026-08-11} }
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How this summary was made. Every section above restates what the published abstract of PMID 26130237 reports — the question, the design, the numbers, the authors’ own conclusion and their stated limitations. We do not add claims the paper did not make, and we keep negative and no-effect findings in. Magellan’s evidence grades are set from research like this and never from affiliate commissions.

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Educational information, not medical advice. This is a plain-English summary of published research; it is not a treatment recommendation and nothing here is intended to diagnose, treat, cure, or prevent any disease. Individual studies can be wrong, and a single paper rarely settles a question. Talk to your physician before acting on any research, especially if you are pregnant, nursing, or taking medication.